Did you not hear about the completely legit and not at all biased research carried out by Dr Fauxci’s great-niece? Fed 20 ferrets LD50 doses of dilithium crystals, 11 died of all causes. Fed 20 ferrets doses of the great new $555 per pill RunDeathIsNear and a mere 9 ** died. That’s eleventy percent more effective.
** Not counting the three that sadly had rare blood clots and had to be put down.
Perhaps between us we could get on the Jimmy Dore show?
ha ha, Jimmy talks way too fast for me to comprehend so I’d just sit there like a lump unfortunately.
The RunDeathIsNear thing was mentioned in the RFK Jr book, specifically cited as being a nickname used by hospital nurses. (Page 157)
Just on the initial topic of the Grundy study - yes it seems a long while back (about 3 weeks).
Twitter now says American Heart Association science article is “unsafe link” because it shows that mRNA vaccines cause heart damage
Link: Twitter now says American Heart Association science article is "unsafe link" because it shows that mRNA vaccines cause heart damage
Twitter’s various reasons for blocking this study are absurd and mindless (that’s probably literally true 
).
The thing that jumps out as the real reason is “We didn’t like what it said”.
We know that anyway, but nice to see it as the only possble way the excuses can mean anything at all.
Citing the article, Twitter says:
“Warning: this link may be unsafe.”
Users are then urged to click the “Back to previous page” button to avoid accessing the article, though there is also a tiny link at the bottom that says, “Ignore this warning and continue.”
To be fair the AHA is backtracking, using the excuse of poor typography.
These surgeons that spend too much time treating critically ill patients when they could be jumping through hoops for corrupt medical and non-medical opposition…
Any excuse for Twitter…back to the article:
"Twitter jumped all over this and took the opportunity to further claim erroneously that the study itself is “potentially spammy or unsafe” and could contain “malicious links that could steal personal information or harm electronic devices.”
Twitter further insisted that the study could “mislead people or disrupt their experience” because it supposedly contains “violent or misleading content that could lead to real-world harm.”
“[C]ertain categories of content that, if posted directly to Twitter, are a violation of the Twitter Rules” was also slapped across the study link to try to deter users from viewing it.
Which of these Twitter believes apply to the Circulation study remains unclear, though it is possible that employees at the social media giant consider all of them to be applicable to its contents. "
THIS AMERICAN HEART ASSOCIATION STUDY MIGHT STEAL YOUR DATA OR HARM YOUR DEVICES!!
Nothing could be more revealing of Twitters true motivation - it doesn’t care if it’s reasons are genuine, or even make logical sense, as long as it deflects from the substance of the paper.
@CJ
Maclom Kendrick here with Dr Cremola. No references, unusually for Dr M - probably because the material is from the above Kendrick’s book. But interesting stuff, off-mainstream. Have you had a chance to read the book yet? Do you have an appointment to have your Glycocalyx repaired? 
ED
Blood Clots May Be the Root Cause of All Heart Disease
Analysis by Dr Mercola
https://articles.mercola.com/sites/articles/archive/2022/02/13/root-cause-of-all-heart-disease.aspx
(Link will vanish in 38 hours due to GIC pressure, but pasted below)
Story at-a-glance
• The thrombogenic hypothesis asserts that blood clotting is the basic underlying pathological process that causes all heart disease
• When a blood clot forms on your artery wall, it will typically be covered over and broken down. A problem arises, however, when the blood clot is not fully eliminated and becomes a ‘vulnerable’ point, and another blood clot forms at the same point. Over time this grows and becomes what’s conventionally referred to as atherosclerotic plaque
• A clot will form where endothelial cells have been stripped away, or are seriously damaged The blood clot will then be covered over by endothelial progenitor cells, which float around in your blood stream at all times. When progenitor cells find an area of damage, where a blood clot has formed, they attach themselves to that area, creating the new endothelial layer. This repair process can gradually create a thickening inside the artery wall itself
• In almost everyone, the process of endothelial damage and blood clotting is an ongoing process. Which means that problems only occur when the damage/blood clotting process occurs faster than the repair process, at which point you will end up with plaque buildup. This thickens the arterial wall, forcing blood flow through a narrower gap. When a large blood clot forms on top of an existing plaque, in this already narrowed area, you can end up with a heart attack or stroke
• Common causes of endothelial damage include such things as viral infections, high blood sugar levels, smoking, diabetes, heavy metals such as lead and aluminum, and high blood pressure
Discussion
In this interview, repeat guest Dr. Malcolm Kendrick, a board-certified family physician and author of the book, “The Clot Thickens: The Enduring Mystery of Heart Disease,” reviews the underlying mechanisms for heart disease, which for the last century has been the leading cause of death in the U.S.
Of all the books he’s written, this is my favorite, as it goes into great detail, giving you the biological understanding of the process of atherosclerosis leading to heart attacks and strokes. He also has solid strategies for lowering your cardiovascular disease risk.
Incidentally, once you understand the disease process, then you can also understand how both COVID-19 and the COVID jab can contribute to heart disease. When asked why he’s taken such an interest in heart disease, Kendrick replies:
“When I was training as a student in medicine, Scotland had the highest rate of heart disease in the world. Early on the answer for why was, ‘Oh, well, it’s because we have such terrible diet, and we eat rubbish food like deep fried Mars bars.’
So, you eat too much saturated fat, the saturated fat gets turned into cholesterol in your bloodstream, and then it’s absorbed into arteries and forms narrowings and thickenings, which all sounds plausible if you don’t think about it too hard.
But I also happen to go to France quite a lot, and what I noticed about France was, they eat a lot of saturated fat. They eat more, in fact, than anyone else in Europe, and certainly more than Scotland. So, [this saturated fat] hypothesis certainly didn’t work for the French. They have the highest saturated fat intake in Europe and lowest rate of heart disease, and this has been the case for decades.
If you took all the risk factors for France and Scotland [such as smoking, high blood pressure and diabetes], then the French had slightly [higher risk], according to conventional thinking. But, in fact, they had one-fifth [the rate among age-matched men].
So, I thought, this is interesting. It doesn’t make much sense according to what we’re told. Then while I was in medical school, a tutor in cardiology said … LDL cannot cross the endothelium. At the time, I didn’t know what LDL was, nor did I know what the endothelium was, but it sounded important.
She had been looking at heart disease as a different process for decades … So, I think that’s really where I got started. Once you start questioning what the problem is, you end up questioning more and more and you start thinking, gosh, this is just nonsense, isn’t it? This whole hypothesis is just nonsense. So, I started picking it apart.”
“The Clot Thickens” is Kendrick’s effort to explain an alternative hypothesis for what actually causes heart disease. If it’s not saturated fat and cholesterol, what is it? In 1852, a Viennese researcher, Karl von Rokitansky, developed what he called the encrustation hypothesis of heart disease.
Today, this hypothesis has been renamed the thrombogenic hypothesis. ‘Thrombo’ stands for thrombosis, i.e., blood clots, and ‘genesis’ means the cause of, or the start of. So, the thrombogenic hypothesis is that blood clots are the basic pathology that causes all heart disease.
We know blood clots cause the final event in cardiovascular disease. We know blood clots cause plaques to grow. Why won’t you accept that blood clots are the thing that starts heart disease in the first place? Because then we have one process all the way through, and it makes sense, because it fits with what you can see. ~ Dr. Malcolm Kendrick
In a nutshell, when a blood clot forms on your artery wall, which can happen for a number of reasons, it will typically be covered over and dissolved. A problem arises, however, if the blood clot is not fully eliminated and another blood clot forms in the same ‘vulnerable’ area. This then becomes what’s conventionally referred to as atherosclerotic plaque.
“The atherosclerotic plaque is basically a buildup of blood clot, repair, blood clot, repair, blood clot, repair,” Kendrick explains. “If the blood clotting process is faster than the repair process, you have a plaque that gradually grows and eventually thickens the artery wall until it narrows sufficiently that the final blood clot, on top of the existing plaque, is the thing that can cause a heart attack or stroke …
If you cut through the plaque and look at it, it almost looks like tree rings. You can see there’s been a clot, repair, clot, repair, clot, repair, clock, repair over the years.
It’s widely accepted that a blood clot forming on an existing plaque will cause the plaque to grow in size. You can find 10,000 papers saying that this is the case. What the mainstream won’t accept is that a blood clot on a healthy artery wall can initiate the whole process.
So, to an extent, all I’m saying to people is, well, we know blood clots cause the final event. We know blood clots cause plaques to grow. Why won’t you accept that blood clots are the thing that starts it in the first place? Because then we have one process all the way through, and it makes sense, because it fits with what you can see.”
As noted by Kendrick, the conventional view is that low-density lipoprotein or LDL gets into the artery wall where it initiates plaque formation. It then, inexplicably, stops initiating plaque, and the plaque continues to grow through the addition of repeated clots.
However, Kendrick says, once you start drilling down into the cholesterol, aka LDL hypothesis, the whole thing starts to fall apart. LDL simply cannot explain the disease progression. Yet despite the many holes in the theory, the idea that LDL causes heart disease is touted as an absolute, indisputable fact.
In order to justify a hypothesis, you need to have a mechanism of action. Once you understand the mechanism of the actual disease process, then you can put the puzzle pieces together. Kendrick begins his explanation:
“Your blood vessels are lined with endothelial cells, a bit like tiles on a wall. Endothelial cells are also covered themselves in a thing called glycocalyx. If you try to pick up a fish, it’ll slip through your fingers; it’s very slippery. The reason it’s slippery is because it’s covered in glycocalyx and the glycocalyx is incredibly slippery. It’s nature’s Teflon.
So basically, in our case, the glycocalyx [is inside] our blood vessels, to allow the blood to travel through without it sticking, without damage occurring. So, you have this kind of damage-repellent layer on top of your endothelial cells.
Now, if that layer is damaged, and then the endothelial cell itself underneath is damaged, then the body will say, ‘Oh, we’ve got damage to a blood vessel, we must have a blood clot there because we could bleed out.’ So, a blood clot forms on the area of damage, and immediately stops [the bleeding].”
The blood clot doesn’t just keep on growing and growing. If it did, you’d die anytime you had a blood clot. Instead, when a clot forms, other processes step in to prevent it getting too big, which is why every blood clot doesn’t cause a stroke or heart attack. Once the clot has stabilized, and has been shaved down, the area is covered over by endothelial progenitor cells, made in the bone marrow, that float around in your blood stream.
When a progenitor cell finds an area that has been damaged, it attaches itself to that area, along with others, forming a new endothelial layer. The remaining blood clot is now lying ‘within’ the artery wall itself. So, basically, it’s the repair process that can lead to plaque buildup within the artery wall. In time, if damage outstrips repair, this can narrow the artery and reduce blood flow.
The question is, what can damage the endothelium in the first place? Here, Kendrick uses the SARS-CoV-2 mechanism as an example:
“The COVID virus enters endothelial cells through the ACE2 receptor. It prefers endothelial cells because they’ve got ACE2 receptors on them. It gets into the endothelial cell and starts replicating, then bursts out, damaging the cell. Bingo, you’ve got an area of damage.
Of course, added to this, when cells have viruses within them, they send out distress signals to the immune system saying, ‘I’ve been infected, come and kill me,’ and so the immune system starts to have a go at the endothelial cells. This is why you can get a problem, because the endothelial cells are being damaged and stripped off.
Blood clotting occurs at the points of damage and hey, presto, you’re having clotting, you’re having strokes, you’re having heart attacks, which is the thing that people at first couldn’t understand [about COVID-19]. Yet it’s very clear that what’s happening is you’ve got damage to the endothelial cells.
Obviously, you and I both know that if you get a [COVID jab], the cells are triggered to produce the spike protein, and these cells are sending out distress messages saying, ‘I’m infected.’ You have to be very careful if you want to stick something into cells that then says to the immune system, ‘Please come and destroy me,’ because that’s what the immune system is going to do.
But moving on from that, what other thing can cause endothelial damage? The answer is things like smoking. Smoke particles get out of your lungs, they go into your blood vessels and they cause damage … You smoke one cigarette and a whole bunch of microparticles appear in your bloodstream, which means endothelial cells are dying.
Luckily as endothelial cells die, another message is sent to the bone marrow saying, we need more endothelial cells and it stimulates endothelial progenitor cell production. These endothelial progenitor cells rush around covering over the areas of damage.
Some smokers have enough repair going on and when you’re younger, it’s okay. As you get older and your repair systems begin to fail a bit, cigarette smoking becomes more and more of a problem.”
Other things that can cause endothelial damage include:
• High blood sugar levels and diabetes. The protective glycocalyx layer is made of proteins and sugars — High blood sugar damages the glycoprotein layer, thinning it down in a measurable way. High blood sugar can reduce the glycocalyx layer by as much as two-thirds. This, in turn, exposes the endothelial cells to the bloods and anything else damaging that might be there.
The damage to the glycocalyx is why diabetics are prone to both arterial and capillary (small vessel) disease. You can’t get atherosclerosis in the capillaries, as there’s no room. Instead, the capillaries become broken down and destroyed. This in turn can cause ulcers, due to poor circulation in the skin of your legs and feet.
Peripheral neuropathy as the ends of nerve cells are deprived of oxygen. Also visual problems (diabetic retinal damage) and kidney damage. Blood pressure may also become elevated as your heart has to work harder to push blood through a network of damaged/missing small blood vessels.
• Heavy metals such as aluminum and lead.
• High blood pressure, as it puts stress on the endothelium — Atherosclerotic plaques (atherosclerosis) doesn’t occur unless the pressure is raised, adding biomechanical stress.
As explained by Kendrick, the glycocalyx layer resembles a lawn, with slippery filaments that stick up. Within this glycocalyx layer you have nitric oxide synthase (NOS), which produces nitric oxide (NO), and you have NO itself, as well as a number of other anticoagulant proteins. The glycocalyx is actually a potent anticoagulant layer, so it stops blood clots forming. If glycocalyx is damaged, your risk of blood clotting increases.
“It’s a very complicated layer,” Kendrick says. “It’s like a jungle full of things that say, ‘Don’t stick to this, stay away from this.’” Within it, you also have albumin, protein complex produced by the liver. Albumin contains the proteins that help maintain and repair the glycocalyx. A fact that most doctors are unaware of is that, if you have a low albumin level, you’re significantly more likely to die of heart disease.
The good news is that while the glycocalyx layer can be rapidly destroyed, it can also be rapidly repaired. (Experiments have shown that in an area where the glycocalyx has been completely stripped off, it can be completely repaired in a single second.) Supplements like chondroitin sulfate and methylsulfonylmethane (MSM) can be helpful in this regard.
“If you try and explain that through the LDL mechanism, it just doesn’t work,” Kendrick says. “They have discovered that if you give chondroitin sulfate as a supplement — which normally is for arthritis and stuff like that — it reduces the risk of heart disease quite considerably. How do you explain that? Well, you can explain that because you’re protecting your glycocalyx.
These are the sort of things that make no sense if you like looking at the conventional ideas of heart disease, but are immediately and easily explained if you say, ‘We have to keep our glycocalyx healthy and we have to keep our endothelial cells underneath them healthy.
Otherwise they will be damaged and stripped off, and then we will get a blood clot, and if we keep getting blood clots at that point, we will end up with a plaque and eventually one of the blood clots on that plaque will kill you from a heart attack or a stroke.”
A lifestyle strategy that can help repair endothelial damage is blood flow restriction (BFR) training. In response to BFR, your body produces vascular endothelial growth factor (VEGF), which acts as “fertilizer” for the endothelium. You can learn the ins and outs of BFR in my free BFR report. VEGF also induces the synthesis of nitric oxide (NO), a potent vasodilator, and it stimulates endothelial progenitor cells.
“NO protects the endothelium. It is anticoagulant — the most potent anticoagulant we have in the body. It’s really the magic molecule for cardiovascular health,” Kendrick says.
“At one time NO was known as Endothelial Derived Relaxation Factor (EDRF) NO was something no one believed could possibly exist in the human body. NO is actually a free radical. Everyone says free radicals are terribly damaging and unhealthy.
To that I reply, ‘Well, you may wish to know that the chemical that is the single most important protective chemical in the body for the cardiovascular system is an incredibly free radical called nitric oxide.”
Some anticancer drugs are designed to block VEGF, as the tumor needs angiogenesis — which is the creation of new blood vessels that are required to provide sufficient ‘nutrients’ Without these new blood vessels, the tumor dies off. Unfortunately, if you block VEGF, you also block NO, which then raises your risk for heart disease.
“These drugs were almost removed from the market,” Kendrick says*, “because despite their anticancer activity, they were procardiovascular disease to quite a scary degree.*
[That’s why], if you are given bevacizumab or Avastin as an anticancer drug, they now give you angiotensin converting enzyme inhibitors (ACE inhibitors), which are blood pressure lowering tablets, and ACE inhibitors have a specific impact on bradykinin, which increases NO synthesis.”
In his book, “The Clot Thickens: The Enduring Mystery of Heart Disease,” Kendrick reviews many different strategies that can lower your disease risk. Here’s a short-list of examples covered in far greater depth in the book, as well as some of my own recommendations that I bring up in the interview:
Avoid unnecessary use of nonsteroidal anti-inflammatories (NSAIDs) such as ibuprofen, aspirin and naproxen — While they effectively inhibit inflammation, they can cause platelet aggregation by blocking COX-2. In other words, they activate your blood clotting system, making blood clots more likely.
Get plenty of sensible sun exposure — Sun exposure triggers NO that helps dilate your blood vessels, lowering your blood pressure. NO also protects your endothelium, and increases mitochondrial melatonin to improve cellular energy production.
Avoid seed oils and processed foods — Seed oils are a primary source of the omega-6 fat called linoleic acid (LA), which I believe may be far more harmful than sugar. Excessive intake is associated with most all chronic diseases, including high blood pressure, obesity, insulin resistance and diabetes.
LA gets embedded in your cell membranes, causing oxidative stress, and can remain there for up to seven years. Oxidative linoleic acid metabolites (OXLAMs) are what’s causing the primary damage, including endothelial damage.
Lower your insulin and blood sugar levels — Simple strategies to accomplish this include time-restricted eating, eating a diet high in healthy fats and low in refined carbohydrates, significantly restricting your LA intake and getting regular exercise.
Address chronic stress, which raises both blood sugar and blood pressure, promotes blood clotting and impairs your repair systems. Cortisol, a key stress hormone, reduces endothelial cell production.
Quit smoking.
https://articles.mercola.com/sites/articles/archive/2022/02/13/root-cause-of-all-heart-disease.aspx
Thanks for the reminder on all this, E! Crucial stuff. I get Joe’s mailouts too, but I’d overlooked this material.
Would a decent tl:dr; be that fats are not so different to a lubricant in any other sealed system?
See this on PHS stats changes:
h/t Eugyppius
Also I watched Dr John last night on a paper about myocarditis deaths and high rates in young men that was very detailed and important “proof” that the risk from vaxing is at least 85 times above background risk, for serious cases. The number of less serious or undetected cases would be at least 10 or 50 times this.
Snap!
Similar data in Australia: Covid-19 Deaths are at record levels in Australia and 4 in every 5 of them are among the Fully Vaccinated – The Expose
Unless there’s a plan to suppress the data in other countries too, you’d think they would be better off trying to explain how the data should be intepreted.
Is that Dr John Campbell, Dimac
Hi @Evvy_dense , still only skimmed it , light on covid and nothing on jabs as far as I recall. I hope to get back to it soon, I will not be going anywhere near the test, label, wait and die process of the NHS! If I step too close to their systems I will be stamped with “old unjabbed git, do not resuscitate, suggest earliest treatment with medazolam !”
cheers
Precisely my sentiments, CJ. Bad situation when hospitals become places to avoid, for danger of being murdered! I remember keenly John O’Looney’s account of the pressure he suffered to take what he happened to know was a dangerous drug, and how he had to be rescued from the hospital - sic! - by his friends. Astonishing. The destructive subversion of our NHS by the gics and their useful idiots is well advanced. Something they’ve been aiming for since 1948.
Likewise. As for the NHS it has become a mystifying mishmash of accounting entities billing each other for no one knows what and with no one taking responsibility for anything. Just like Obamacare and soon to morph into exactly that.
tl:dr; the usual mafia accumulate more baksheesh
The reporting systems will be deemed unfit for purpose and replaced with something more agreeable to Big Harma. I believe the Scotland stats are being suppressed because Bad Actors use them for DisMisMalInformation.
File Under: If the evidence doesn’t fit the theory get new evidence.
EDIT: I see Evvy has this covered at
Oh it has been seen, the same crew (in their younger incarnations), were responsible for AIDS R!
Again R would you choose a category (Covid), for your posts please? Reason being that in order to subvert Twitter (I have found), that simply pointing people to the Covid category here (with some other “generic” pointers), does not attract censure. Specifying does! So #Covid category o.k! See?
Well I never saw that! One point I’d make is that the comparison of the number of deaths with the “first wave”, or second wave around August last year, is not very valid, as the number of infections then was minimal. It’s only with the coming of Omicron that cases around the country suddenly got into the hundreds of thousands a day, even though for quite a short period, and are now back to ten thousand only. What actually happened was that the number of deaths associated with Omicron fell to near zero, while deaths associated with Delta made up the bulk of deaths in all states. They stopped doing analyses on the genotype, so it’s speculation. It is also true that many of the “cases” have been in younger people - I think 50% below the age of thirty. Many of these wouldn’t have been tested before, though the virus really wasn’t circulating then, What IS the case however, is that most of the hospitalisations and deaths are in fully vaxed people, and some of these may be due to vaccine injury. Even when it is admitted that a patient had other serious health issues, if they tested positive entering hospital that is what killed them.
And the latest? a poster at a rally says it all - “Just one more jab and you’ll be free!” Andrews says that quite soon a booster may be necessary for the jabbed to enter a bar or restaurant, because winter will be here soon and there could be another wave.
@Jamie . I too find it impossible to converse with the blind. I’m not sure where to find it, but there was one doctor’s 20 year study of jabbed versus unjabbed (any vaccines) and the outcomes for things like ADHD, obesity, asthma, ecezma, etc and it was plain for any fool (even the blind) to see the difference. The analyses was for something like 1,200 kids. If you can find it, print out and just give them a copy and walk away! Evvy may well know where to get a copy.
Hi PatB. Is this the one
by James Lyons-Weiler 1,* and Paul Thomas 2 1
The Institute for Pure and Applied Knowledge, Pittsburgh, PA 15101, USA2
Integrative Pediatrics, Portland, OR 97225, USA*
Author to whom correspondence should be addressed.
Int. J. Environ. Res. Public Health 2020, 17(22), 8674; IJERPH | Free Full-Text | Relative Incidence of Office Visits and Cumulative Rates of Billed Diagnoses Along the Axis of Vaccination
Received: 23 October 2020 / Revised: 14 November 2020 / Accepted: 18 November 2020 / Published: 22 November 2020
(This article belongs to the Section Children’s Health) View Full-Text Download PDF Browse Figures
Citation Export
We performed a retrospective analysis spanning ten years of pediatric practice focused on patients with variable vaccination born into a practice, presenting a unique opportunity to study the effects of variable vaccination on outcomes. The average total incidence of billed office visits per outcome related to the outcomes were compared across groups (Relative Incidence of Office Visit (RIOV)). RIOV is shown to be more powerful than odds ratio of diagnoses. Full cohort, cumulative incidence analyses, matched for days of care, and matched for family history analyses were conducted across quantiles of vaccine uptake. Increased office visits related to many diagnoses were robust to days-ofcare- matched analyses, family history, gender block, age block, and false discovery risk. Many outcomes had high RIOV odds ratios after matching for days-of-care (e.g., anemia (6.334), asthma (3.496), allergic rhinitis (6.479), and sinusitis (3.529), all significant under the Z-test). Developmental disorders were determined to be difficult to study due to extremely low prevalence in the practice, potentially attributable to high rates of vaccine cessation upon adverse events and family history of autoimmunity. Remarkably, zero of the 561 unvaccinated patients in the study had attention deficit hyperactivity disorder (ADHD) compared to 5.3% of the (partially and fully) vaccinated. The implications of these results for the net public health effects of whole population vaccination and with respect for informed consent on human health are compelling. Our results give agency to calls for research conducted by individuals who are independent of any funding sources related to the vaccine industry. While the low rates of developmental disorders prevented sufficiently powered hypothesis testing, it is notable that the overall rate of autism spectrum disorder (0.361%) in the cohort is one-fifth that of the US national rate (1.851%). The practice-wide rate of ADHD was roughly half of the national rate. The data indicate that unvaccinated children in the practice are not unhealthier than the vaccinated and indeed the overall results may indicate that the unvaccinated pediatric patients in this practice are healthier overall than the vaccinated. View Full-Text
Here’s a thumb-steadier:
Children’s Health Defense (CHD) has assembled nearly 60 studies that find vaccinated cohorts to be far sicker than their unvaccinated peers.
Thanks @Evvy_dense . I suggested giving the true Covid believers a flyer and walking away. Looking at the PDF, you may have to make that a folder, but even those deaf, dumb and blind, can’t disbelieve the charts, can they? I wonder what they’d make of them over at TLN?
“I wonder what they’d make of them over at TLN?”
“That’s just a big flyer - I need sources. Links! References!! Gullible anti-vaxxers…Oh you’ve got these as well - Jesus, you don’t expect me to read all that!”